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Cardiogenic shock is characterized by a profound decrease in cardiac function that results in sustained hypotension or need for vasopressors and leads to end-organ hypoperfusion. It carries a nearly 50% early mortality, depending on the underlying cause.
Leading causes of cardiogenic shock have shifted from infarct-related events to acute-on-chronic heart failure and nonischemic etiologies, including myocarditis, valvular or pericardial disease, and cardiac arrest (see ).
As clinicians now know, immediate revascularization in infarct-related cardiogenic shock reduces mortality. Current evidence supports revascularizing only the culprit lesion in the acute setting for patients with multivessel coronary artery disease.
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